© 2000 by European Society of Cardiology
Copyright © 2000, European Society of Cardiology
Chemokines and cardiovascular diseases
Department of Cardiovascular Medicine, Kyoto University Graduate School of Medicine, 54 Kawahara-cho, Shogoin, Sakyo-ku, Kyoto 606-8507, Japan
* Corresponding author. Tel.: +81-75-751-3185; fax: +81-75-752-0856 sasayama@kuhp.kyoto-u.ac.jp
Received 22 October 1999; accepted 22 October 1999
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See article by Damås et al. [16] (pages 428–436) in this issue.
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1 Introduction |
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Recruitment of circulating leukocytes requires a variety of soluble and cell-associated factors, which mediate their migration into injured or inflamed tissues. Chemokines (chemotactic cytokines) are low-molecular-weight (8–10 kiloDaltons) proteins characterized by their ability to induce directional migration of leukocytes, thought to play important roles in controlling inflammation and immune responses [1,2]. Chemokines mediate not only the migration but also the growth and activation of leukocytes and other cells. They are produced by a variety of cell types of hematopoietic and nonhematopoietic origin in response to antigens, polyclonal stimulants, cell irritants, and cytokines, and their biologic effects are mediated by the
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2 Chemokines and atherosclerosis |
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3 Chemokines and myocardial ischemia |
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4 Chemokines and heart failure |
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