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Cardiovascular Research 1999 43(4):830-831; doi:10.1016/S0008-6363(99)00192-3
© 1999 by European Society of Cardiology
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Copyright © 1999, European Society of Cardiology

Are the arrhythmias due to myocardial ischaemia and infarction dependent upon the sympathetic system?

M.K Pugsleya, M.J.A Walkerb,* and S.L Yongb

aDepartment of Pharmacology & Toxicology, XOMA (US) LLC, Berkeley, CA, 94710, USA
bDepartment of Pharmacology and Therapeutics, Faculty of Medicine, The University of British Columbia, 2176 Health Sciences Mall, Vancouver, B.C., Canada V6T IZ3

* Corresponding author. Tel.: +1-604-822-9531; fax: +1-604-822-9578 rsdaa@interchange.ubc.ca

Received 3 June 1999; accepted 3 June 1999

The first 10% of the full text of this article appears below.

See article by Du et al. ([10], pages 919–929) in this issue.

Many studies have implicated the sympathetic nervous system as being of importance in the genesis of the arrhythmias induced by myocardial ischaemia and infarction [1]. This can involve increased activity in the sympathetic system or the local release of norepinephrine from sympathetic nerve endings exposed to the extracellular conditions that occur in ischaemic tissue. Evidence to support the role of the sympathetic nervous system has included the use of drugs to modify the system or extirpation of nerves, but rarely mimics or increases sympathetic activity. It has to be remembered that, regardless of the manner in which evidence is accumulated, activation of . . . [Full Text of this Article]


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