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Cardiovascular Research 1999 43(2):282-284; doi:10.1016/S0008-6363(99)00165-0
© 1999 by European Society of Cardiology
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Copyright © 1999, European Society of Cardiology

Sarcoplasmic reticulum Ca2+ ATPase gene expression to the rescue of myocardial contractility in hypothyroid associated heart failure

Carlo Venturaa,b,*, Gianfranco Pintusa,b and Margherita Maiolia,b

aDepartment of Biomedical Sciences, Division of Biochemistry, Laboratory of Cardiovascular Research, University of Sassari, Viale San Pietro 43/B, 07100 Sassari, Italy
bNational Laboratory of the National Institute of Biostructures and Biosystems, 07033 Osilo, Italy

* Corresponding author

Received 22 April 1999; accepted 22 April 1999

The first 10% of the full text of this article appears below.

See article by Bluhm et al. [18] (pages 382–388) in this issue.

Recent advances in animal genetics and transgenic technology have blossomed into one of the dominant strategies to explore the consequences of gene defects in different organ systems in the in vivo context. Such a perspective has been progressively applied to the cardiovascular system to dissect complex in vivo physiologic and pathologic states. The encouraging results achieved so far in the field of heart hypertrophy and failure with the aid of engineered mouse models have even suggested consideration for the cardiovascular system as a paradigm for other complex apparata, including the pulmonary, renal and neural systems [1].

It is now becoming increasingly evident . . . [Full Text of this Article]


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